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The higher plasminogen activator inhibitor type 1 levels and lower urokinase plasminogen activator levels in umbilical cord of these patients are suggestive of decreased fibrinolysis in the fetal circulation.
Increased basal tissue concentration of MELM was seen in the spinal cord of these animals [37] and this was associated with a clear reduction of release [38].
Analysis of cHes6-1 expressinn in the spinal cord of these embryos shows that the number of cells expressing cHes6-1 is reduced when Notch is ectopically activated and neurogenesis is blocked (Fig. 5D-Fi).
Analysis of cHes6-1 expressinn in the spinal cord of these embryos shows that the number of cells expressing cHes6-1 increasedsed when neurogenesis is accelerated due to Notch inactivation (Fig. 2N, 5G-Ii).
This is consistent with the identification of TDP-43 positive inclusions in the brain and cord of these mice.
In addition, isolated mitochondria from the spinal cord of these mutant SOD1 mice showed decreased Ca2+ buffering capacity [ 8], and dysfunction of oxidative phosphorylation and consequently decreased ATP synthesis [ 9, 10].
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The color of the cords of these drums also indicates the skill of the musician: Orange and red for amateur players, light blue for performers with expertise, and lilac for masters of the instrument.
The bone marrow and spinal cords of these rats were taken out for formaldehyde measurement with HPLC.
However, no significant lesions were seen in the spinal cords of these animals.
These results suggest an increase in the number of FoxP3 expressing cells in the spinal cords of these mice.
This finding indicates that the higher levels of total SOD1 in spinal cords of these animals stimulate the generation of these aggregates.
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