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A partial copy is shown in the Shaanxi Historical Museum in Xi'an.
The structure described here is shown in red, while the second copy is shown in gray.
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An advance copy was shown to The New York Times.
For the comparison of the image quality, the degradation of watermarked images and averaged copies is shown in Figure 5.
The lifetime BMI trajectory of the risk haplotype (carriers, at least one copy) vs. non-risk haplotype (non-carriers, no copies) is shown in Fig. 3a.
A drawing of the copies is shown in figure 2 and represents the integrity of the putatively full-length copies compared with RC.
An example is given in Figure 2, where the distribution of the copy number estimations of the 1 430 trans-sialidase annotated copies is shown together with the distributions for all annotations and all hypothetical genes.
Otherwise, the distributions of the two separate copies were shown (copy 1, copy 2).
Significant differences of viral genome copies were shown between treated and untreated cells at 24, 48 and 72 hpi, which agrees with the results obtained in PK-15 cells (data not shown).
For instance, in human, whereas an elevated number of copies of the β-defensin gene DEFB103 in the East Asia population is associated to increased resistance to influenza [ 42], high α-defensin (DEFA1A3) gene copies were shown to be significantly correlated with susceptibility to severe sepsis [ 43].
For P element repression (P cytotype), the existence of a maternally transmitted cytoplasmic component (pre-P cytotype), coupled with chromosomally inherited P copies, was shown to be necessary to establish strong repression in the zygote (Niemi et al. 2004; Ronsseray et al. 1993).
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com