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According to our analysis, many of the down-regulated genes encode mitochondrial proteins; conversely, the expression levels of genes that encode proteins localized in mitochondria tend to be negatively regulated in the long-lived mutants.
Conversely, the expression levels of HDAC1 mRNA were significantly decreased by 5-Aza-dC treatment.
Conversely, the expression levels of Noggin are directly linked to those of both TDP-43 and Numb).
Conversely, the expression levels of sense transcripts corresponding to up-regulated antisense transcripts were decreased (p = 0.05, Figure 6A).
Conversely, the expression levels of glyceraldehyde-3-phosphate dehydrogenase (GAPDH) and that of another S100 family protein, S100A10, were unaffected by AIRE (Fig 5A).
Conversely, the expression levels of GFAP, S100B, and AQP4 transcripts, representing the astrocyte population, and to a lesser extent those for AIF1, LGALS3, CD68, and EMR1 representing the microglial population, were increased, especially in the temporal cortex and hippocampus.
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Conversely, the expression level of CD36 mRNA dramatically increased (62-fold compared with the CONT group) after 2 weeks of hindlimb suspension in this study.
Conversely, the expression level of acetylated α-tubulin was upregulated significantly in Rpgrip1 nmf247 compared with the wild-type retinas, despite of its strong decrease at the photoreceptor cilium of Rpgrip1 nmf247.
Conversely, IMMP2L_ DOCK4 microdeletions do not affect the expression levels of ZNF277.
The expression levels of Noggin were conversely significantly upregulated after lesion (one-way ANOVA: F2,27 = 5.155, P = 0.013).
Conversely, certain genes were predicted to be potential targets of several miRNAs, suggesting that the effect of miRNAs on the expression levels of these mRNAs may be "redundant".
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