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As shown by Katja Brückner and co-workers in 2011, plasmatocytes proliferate most actively in the sessile subepithelial patches, where cell division appears to be controlled by the peripheral nervous system (Makhijani et al., 2011).
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Moreover, the rod surface chemistry can be controlled by designing the peripheral groups of the lateral mesogens.
The mechanical properties were controlled by the length of the peripheral polystyrene blocks, which were varied from 2.9 to 11.3 kg/mol.
In mammals, body weight and energy homeostasis are controlled by the interplay between the peripheral signals and the brain, particularly the hypothalamus (1).
We found no clear correlation between the clock/functional genes, suggesting that some peripheral functions may be controlled by the SCN without the direct involvement of peripheral clock genes (at least in as far as can be seen from the mRNA level).
Circadian clocks in peripheral organs are controlled by the interplay between transcriptional activators and repressors, which generates a negative feedback loop of core clock gene expression.
Further, previous work suggests that rhythms in Per2/PER2 expression in peripheral tissues are specifically regulated by systemic cues controlled by the SCN [ 29, 48, 49].
This regulation is controlled by the release of hormones (eg, growth hormone, oxytocin, vasopressin) or by the stimulation of peripheral endocrine glands (eg, adrenal cortex, thyroid, gonads).
All of these clocks are controlled by the master pacemaker of the hypothalamic suprachiasmatic nuclei (SCN), which synchronizes all of the body's "peripheral" clocks with the outside world.
Conformation and intermolecular associations can be controlled by peripheral functionalization leading to optimization of crystallization length scales.
BAT function is controlled by both the central and the peripheral nervous system.
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