Sentence examples for control smokers from inspiring English sources

Exact(32)

This preliminary study used a within-subject, placebo-controlled design in smokers with schizophrenia (n = 6) and healthy control smokers (n = 8) to examine the effects of MEC (10 mg/day) on ad-lib smoking behavior, topography, nicotine levels, and tobacco craving across two smoking deprivation conditions (no deprivation and 12-h deprivation).

MEC also increased tobacco craving in smokers with schizophrenia but not in control smokers.

However, no allelic imbalance was detected between CH cases and control smokers at any of the tested polymorphic variants (including the functional non-synonymous SNP rs16969968) (Additional file 1: Table S3).

The result showed that the screening model (including miR-378a-3p miR-378a-3p miR-378a-3p and miR-379-5p) distinguishes between patients with any kind of nodules and control smokers, with andarea miR-200-5p miR-200-5p miR-200-5practeristic curve (AUC) of 0.908, sensitivity of 97.5% andistinguishes of 72.0%.

Applying a double-blind, placebo-controlled cross-over design, the effects of 1 mg nasal nicotine on brain activity elicited by a visual oddball-type task in N=14 schizophrenia and N=15 control smokers were studied with simultaneous EEG-fMRI.

Since the insula is implicated in nicotine dependence, we hypothesized that SZ smokers will have greater dysfunction in smoking-related insular and dACC circuits than normal control smokers (NC) independent of smoking severity, consistent with an inherent disease-related weakening of smoking-related circuits.

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Similar(28)

Only the lung cancer and control smoker cohorts were used for this analysis.

For each subject in the control smoker and lung cancer cohorts, a lung cancer susceptibility score was derived according to these variables and their distributions compared [27], [32].

Several genes are involved in the pathogenesis of CD, and in this study the gene expression differences of the descending colonic mucosa were investigated in CD (smokers or never smokers) and controls (smokers or never smokers).

In summary, we profiled the global serum miRNA expression in nonsmoking controls, smokers, and lung-cancer patients.

Comparisons of the serum miRNA expression levels in the nonsmoker controls, smokers, and lung-cancer patients were made with Kruskal-Wallis one-way ANOVA.

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