Epigenetic modifications control cell proliferation, differentiation, migration, and so on.
There has long been a search for a therapeutic agent to inhibit or control cell proliferation.
DUBs have amongst others been implicated in cellular signaling pathways that control cell proliferation and differentiation.
SCF complexes control cell proliferation through degradation of critical regulators such as cyclins, CDK inhibitors and transcription factors.
Complex developmental programs require orchestration of intrinsic and extrinsic signals to control cell proliferation, differentiation, and survival.
MiRNAs can cause degradation or translational repression of mRNA to negatively regulate more than 30% of the genes to control cell proliferation, inflammation, and metabolism.
The expression of OsARGOS in Arabidopsis may activate the signaling pathways that control cell proliferation and cell expansion during the course of plant growth and development.
Based on these results, we suggest that the RhoA-ROCK-PTEN pathway acts as a molecular switch to control cell proliferation and determine anchorage dependence.
An increasing demand for new strategies in cancer prevention and regenerative medicine requires a better understanding of molecular mechanisms that control cell proliferation in tissue-specific manner.
Unique spatial and temporal patterns of expression within these tissues suggest that MELK plays a prominent role in cell cycle control, cell proliferation, apoptosis, cell migration, cell renewal, embryogenesis, oncogenesis, and cancer treatment resistance and recurrence.
We conclude that they differentially control cell proliferation in dorsal and ventral iris.
