Sentence examples for contributions to pathogenesis of from inspiring English sources

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The data we present here is the first comprehensive analysis of the vsp gene family from the Genotype A1 WB isolate with an emphasis on vsp characterization, function, evolution and contributions to pathogenesis of this important pathogen.

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Here, we tested in prevalent CA-MRSA strains whether PVL regulates global gene networks and evaluated its contribution to pathogenesis of bacteremia, a hallmark feature of invasive S. aureus disease and the most prevalent clinical syndrome of invasive CA-MRSA disease in particular [19].

We aim to investigate the expression patterns of miR-1246 in B cells and its contribution to pathogenesis of SLE.

We conclude that angiotensin and endothelin-receptor activation via anti-AT1R and anti-ETAR Abs mediate pathogenic effects, indicating their contribution to pathogenesis of SSc.

Thus latency of enzymes, the nature of the latency and accessibility of the latent enzymes to activating mechanisms must all be taken into account in any analysis of their contribution to pathogenesis of local lung disease.

Further interpretations of contributions to pathogenesis by FcγRs must be made in the context of LD involving CNV regions.

Isogenic mutants, generated to secrete ExoU, ExoS, or ExoT, were evaluated for their relative contributions to pathogenesis in a mouse model of acute pneumonia [ 50].

Evidence of genetic contributions to the pathogenesis of the disease are based on familial aggregation, predominance in patients with Mediterranean or Asian ancestry and association with the human leukocyte antigen (HLA) B51 (HLA-B51) region in several ethnic groups.

Collectively, these data suggest that SasF and VraE might make contributions to the pathogenesis of systemic inflamation, but not to the development of arthritis.

However, different TLRs provide distinct or synergistic contributions to the pathogenesis of SLE.

Loss of pVHL results in constitutive activation of the transcription factors HIF-1 and HIF-2, whose relative contributions to the pathogenesis of the VHL phenotype have yet to be defined.

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