The AT1R is also known to regulate angiogenesis, migration, and apoptosis all of which could contribute to regulating tumour growth [ 24, 25].
These results show how changes in NE composition contribute to regulating heterochromatin positioning, gene expression, and cellular differentiation during development.
We thus set out to investigate whether miR-24 might contribute to regulating p16 expression during replicative senescence.
As both, EED1 and UME6, are repressed by Nrg1, this mechanism could contribute to regulating hyphal extension.
Our results strongly suggest that OsLIC plays critical roles by influencing development of lateral and intercalary meristems, which contribute to regulating rice plant architecture.
These results suggest that USP10-mediated TBP deubiquitination might also contribute to regulating TBP levels during myogenesis.
Other residues in the C-terminus, as well as other regions of the AQP1 channel could contribute to regulating channel activation.
This mechanism could therefore contribute to regulating translational efficiencies for endogenous Per1, but not for the luciferase reporter, which only carries the Per1 3' UTR.
This indicates that the Argonaute constituent of the miRISC, as well as its interacting proteins, contribute to regulating protein expression by different mechanisms.
Understanding how these more newly identified GLUT isoforms contribute to regulating myocardial glucose transport will enhance our comprehension of the normal physiology and pathophysiology of the heart.
