Web content has been identified as one of the main factors contributing to repeat visits.
In proliferative cells, DNA replication can contribute to repeat instability either by switching the direction of replication, which changes the repeat sequence that serves as the lagging-strand template (origin switching), or by shifting the location of the origin of replication without altering the replication direction (origin shifting).
Social support might contribute to repeat mammography by providing enabling factor [ 32].
If rectal infection is indeed more difficult to treat with azithromycin than cervical infection, then auto-inoculation could contribute to repeat cervical infection in women [ 58– 60].
Mechanisms for this instability have been extensively studied and it turns out that any mechanism involving new DNA synthesis such as replication, recombination and repair can contribute to repeat rearrangements [ 32].
The consensus opinion at present is that several processes, including replication, recombination, DNA repair and transcription, contribute to repeat instability and that the formation of unusual non-B-DNA structures formed by the repeats is at the heart of the expansion processes [ 7, 8].
The underlying process is currently thought to involve the generation of abnormal DNA structures induced by factors such as replication slippage, DNA repair and recombination, that can contribute to repeat instability acting either separately or in combination [ 29- 32] and these mutations underlie pathogenic expansions [ 33] and genetic anticipation [ 34, 35].
To determine the variables that contribute to repeated patient non-compliance with showing up to clinic appointments.
We found the ReHo properties of the temporal pole were positively correlated with the duration of disease, which suggests that temporal pole excitability as sensitization during both the resting-state and stimulation may contribute to repeated migraine attack.
To evaluate phagocytic activity and neutrophil oxidative burst functions in patients with spinal cord injury (SCI) because alterations in neutrophil metabolic activity can be one of the causes of immune mechanism damage contributing to repeated bacterial infections.
