Exact(6)
Experimental results further show that the co-doping contributes to induce the lattice distortion, modify the particle morphology, and increase the electronic conductivity.
This contributes to induce in most patients a high level of anxiety, which may show in a different way according to the gender susceptibility, resulting in depression in females and aggression and irritability in males [ 4].
A recent study showed that rhein contributes to induce apoptosis of human cancer, including lung adenocarcinoma A549, myelogenous leukemia HL-60, lung squamous carcinoma CH27, cervical carcinoma HeLa cells, neuroblastoma IMR-32, bladder cancer T24, and hepatoma HepG2 cells [ 8].
Similar results were found in the study of pathogen resistance responses, where a repression of a number of auxin responsive genes (including genes SAUR, Aux/IAA, auxin importer AUX1, auxin exporter PIN7) were significantly repressed [ 67]; supporting the idea that down-regulation of auxin signaling contributes to induce immune responses in plants [ 68].
On the other hand, a cell-cycle-regulated switch of those ssDNA-binding proteins was needed to re-establish telomere capping after DNA replication, and now it is known that telomeric repeat-containing RNA (TERRA) contributes to induce that switch (Flynn et al. 2011).
In our opinion, these paradoxical data might be reconciled by the hypothesis that miR-21 acts and/or contributes to induce senescence triggered by hyperoncogenic signalling in normal cells similar to oncogenic Ras (Serrano et al., 1997).
Similar(54)
Taken together, our data suggest that miR-21 is induced in normal human cells undergoing replicative or stress-induced senescence in vitro and in vivo and contributes to inducing and/or maintaining the senescence-associated growth arrest.
However, it has been reported that ROS generated by vitamin C and K3 can induce degradation of HSP90AA1, and therefore contributes to inducing cancer cell death [ 12].
A transient increase in AOS expression has been observed in wounded leaves [ 47] and it contributes to induced JA levels [ 48– 50].
We propose that this habituation of short-range inhibition may contribute to induce the interictal lack of habituation of TR-VEP N1-P1 seen in migraine patients.
MIG and IP-10 stimulate the directional migration of activated T cells, thus contributing to induce HIV-specific CTL responses [55].
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