On the other hand, changes in the olefinic bond, although resulting in a slight reduction in the capacity to capture DPPH radicals, and the increase in the hydrophilic character can compensate and contribute as a differential in the antioxidant action.
Several non-biological factors can contribute to a differential identification across different environments.
We hypothesize that corticosteroids contribute to a differential gene expression in the femoral head in rats with early ANFH.
Similarly, patho-physiological conditions like prostacyclin receptor mutations [ 37, 38] contribute to a differential platelet inhibition decreasing the protection against unwanted platelet activation and aggregation.
As fluoroquinolone activity is concentration dependent, these factors may contribute to a differential in a sub-therapeutic exposure among urinary pathogens between moxifloxacin and other fluoroquinolones that are excreted in higher urinary concentrations.
This perspective helps to illuminate those aspects of the adaptive system that contribute to a differential exposure to stimuli and to the coping strategies (resources) that women mobilize against stimuli or problems [ 2, 5, 75].
Based on reported data on corticosteroid effects on the endothelium and their role in apoptosis, we hypothesized that corticosteroids contribute to a differential gene expression in rats with early ANFH.
Because of the random sampling requirement for tests of statistical significance, we must assume that various human populations had an equal likelihood of becoming clinically ill before aerial treatment and that no preexisting factors contributed to a differential in disease experience.
Moreover, induction of lipid catabolism genes in the intestine may be involved in the anti-obesity effect of diacylglycerols as compared with triacylglycerols [ 18, 19] and it may even contribute to a differential sensitivity of two inbred mice strains to an obesogenic high-fat diet [ 20].
However, there are some disparities in the features found in the 5' and 3' endings, namely some N-myristoylation and phosphorylation sites that are not present in all sea bass Slc11a2-β Slc11a2-βd which may also contribute to a diformsntiandpost-transcriptional regulation and subcellular localization.
