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The second part of the study examined whether NE α2A adrenoceptor and/or DA D1 receptor actions contributed to the enhancing effects of MPH on PFC function.
D1 receptors also play a key role in striatal function, and it is possible that these actions outside the PFC also contributed to the enhancing effects of MPH on the delayed alternation task.
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Based on this knowledge, it was important to ensure that neither of these factors contributed to the enhanced invasion observed in our assay.
These results are consistent with α2 adrenoceptor actions contributing to the enhancing effects of oral MPH.
These results are consistent with D1 receptor actions contributing to the enhancing effects of oral MPH.
Both NE α2 adrenoceptor and DA D1 receptor stimulation contribute to the enhancing effects of MPH on working memory in rodents.
The outside barrier layer and the inter-diffused layer could contribute to the enhanced oxidation resistance and the adhension of the coated Zr coupons, which would be highly desirable for the zirconium claddings.
This eliminated the potential that soluble factors in the matrix were being released by the tugging of the beads on the matrix and contributing to the enhanced invasion.
Taken together, these results suggest that any remodeling or release of soluble factors from the matrix due to the movement of the magnetic beads does not contribute to the enhanced invasion we observe upon mechanical stimulation.
225Ac was considered as a suitable cytotoxic agent for coupling to E4G10 because of its four alpha particle emissions per decay of a 225Ac atom, which contributes to the enhanced the potency of 225Ac labeled constructs.
This 1,25-dihydroxyvitamin D3-induced Ca2+-uptake dramatically increases proliferation rate and a number of the cells entering into the S-phase and also contributes to the enhanced apoptosis resistance.
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