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The inability to diagnose PC at an early, localized, and curable stage has contributed to poor prognosis.
A recent report demonstrated that MET gene activation as assessed by fluorescence in situ hybridization (FISH) analysis contributed to poor prognosis in NSCLC patients who received surgical treatments [ 9].
Notably, 75%andd 62.5% of patients who died developed fatal infectious diseases and exacerbation of PAP, respectively, suggesting that the progression of PAP per se and/or PAP-associated infection contributed to poor prognosis.
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However, our present data do not provide an explanation how Axl by itself contribute to poor prognosis.
Angiogenesis contributes to poor prognosis of cancer patients.
These results indicate that high expression of miR-548q and miR-483-5p miR-483-5p miR-483-5poor prognosis.
SELDI biomarker and patterns could complement genetic and molecular heterogeneity which contribute to poor prognosis [ 23].
Impairment of the cardiovascular system contributes to poor prognosis in severe human sepsis.
Even these results are in line with earlier findings and likely to contribute to poor prognosis for clear cell carcinomas.
Several studies demonstrated that the quiescent, non-cycling state of ASCs may contribute to poor prognosis [ 4- 6, 9].
Critical issues contributing to poor prognosis include the inability to detect early stage disease and the capacity of ovarian cancer cells to manipulate the tumor microenvironment [ 2– 4].
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com