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The industrial strains present a genome of more complex constitution with variable number of chromosomes and ploidy that can contribute to adaptation to the process of fermentation in industrial scale (Carreto et al. 2008; Lucena et al. 2007).
Adaptation was not considered dependent on resistance strategy: according to the experts, resistance actions are not needed to implement or to support the effectiveness of actions that contribute to adaptation.
Thus far, no clear or consistent patterns have been revealed, but we do have evidence that urban habitat can shape endocrine traits, and that those traits might contribute to adaptation to the urban environment.
In this work we used a genome-wide screening of S. cerevisiae diploid mutant strain collections to identify genes that potentially contribute to adaptation to low temperature fermentation conditions.
The purpose of this study was to understand the impact of living with Klinefelter syndrome (XXY) as an adolescent or an adult and to examine the factors that contribute to adaptation.
Enlargement of sebaceous glands may also contribute to adaptation to dry, cold conditions.
It is unclear how sleep timing, chronotype, and circadian clock gene variation contribute to adaptation to shift work.
We identified several immediate changes signifying cellular processes that might contribute to adaptation and growth in blood.
Here, we present evidence that chronotype and newly defined sleep strategies contribute to adaptation to shift work.
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However, few pathways such as tetrahydrofolate biosynthesis were found to be variable and could be contributing to adaptation towards virulence such as antibiotic resistance.
Although our model postulates that the induced mutator phenotype contributes to adaptation, it is possible that chronic hypermutability per se does not.
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