Immunosuppression was studied using a nickel contact hypersensitivity recall model.
Popov, A., Mirkov, I. & Kataranovski, M. Inflammatory and immune mechanisms in contact hypersensitivity (CHS) in rats.
Christensen, A.D. & Haase, C. Immunological mechanisms of contact hypersensitivity in mice.
Weber, F. C. et al. Neutrophils are required for both the sensitization and elicitation phase of contact hypersensitivity.
Sawada, Y. et al. Resolvin E1 inhibits dendritic cell migration in the skin and attenuates contact hypersensitivity responses.
Bennett, C.L. et al. Inducible ablation of mouse Langerhans cells diminishes but fails to abrogate contact hypersensitivity.
When urushiol comes in contact with the skin, it initiates the contact hypersensitivity reaction.
In contact hypersensitivity, inflammation occurs when the sensitizing chemical comes in contact with the skin surface.
Streilein, J. W. and P. R. Bergstresser (l988) Genetic basis of ultraviolet-B effects on contact hypersensitivity.
In addition, using the local lymph node assay, a standalone in vivo method for hazard identification of contact hypersensitivity, we showed that colloidal silica nanoparticles act as an immunogenic sensitizer and induce contact hypersensitivity but not MPS nanoparticles.
Honda, T., Egawa, G., Grabbe, S. & Kabashima, K. Update of immune events in the murine contact hypersensitivity model: toward the understanding of allergic contact dermatitis.
