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Table 3 lists Relative Glucose Consumption, Cell Viability and Glucose Consumption Ability of medicinal plants.
The time courses for acarbose accumulation, substrate consumption, cell growth, and impurity C formation in batch culture of A. utahensis ZJB-08196 were firstly investigated.
HIF-1 is a master regulator of cellular adaptation to oxygen deprivation and acts as a survival factor in hypoxic tumor environment, mainly activating the transcription of genes involved in glycolytic metabolism, oxygen consumption, cell migration and invasion [ 15, 32].
First, the entity names (cells, molecules) and process names (oxygen consumption, cell proliferation) are not standardized, which impedes retrieval of the model in a search, and perhaps, the understanding and correct reuse of model components once found (not portable, consistent, retrievable or reusable).
Time courses of acarbose formation, maltose and glucose consumption, cell growth, and impurity C accumulation during batch cultivation of A. utahensis ZJB-08196 in fermentation medium with the addition of validamine at 20 mg/L at the beginning of fermentation were shown in Figure 5. Acarbose titer reached the maximum value of 4950 ± 56 mg/L at 168 h of cultivation.
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To test H2 production and consumption, cells were grown to stationary phase (16 18 h, 37°C) in LBK buffered at the assay pH in closed screw-cap tubes rotating slowly (8 rpm) at 37°C.
Although the GlcNAc consumptions, cell growth, and the ethanol titers of these strains were lower than those at 30 °C, NBRC10063 demonstrated relatively high fermentation ability at 35 °C.
Conditions for optimal growth were determined by measuring substrate consumption and cell dry weight with two different cell lines, flask volumes, and starting inoculum densities.
They also demonstrate that a cessation of oxygen consumption after cell death is beneficial for the remaining cells, since more oxygen becomes available for these cells.
NDI1-NDUFS3-A549 cells weresistantantothethe metformin-mediated reduction in cellular and mitochondrial oxygen consumption and cell proliferation.
She stressed that the study only showed an association and did not prove that sugary drink consumption caused cell ageing.
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