Sentence examples for considerable activation of from inspiring English sources

As shown in Fig. 5., tetracycline induced expression of scNS3 and full NS3-4A has led to considerable activation of the cleavable toxin, while no profound enhancement in cytotoxicity of the control uncleavable toxin was observed.

As shown in Fig. 3., tetracycline induction for expression of scNS3 and full NS3-4A has led to considerable activation of the cleavable toxin which presented only a minor cytotoxic effect against uninduced cells.

The essence of the study indicates that besides considerable activation of the HPA through the restraint conditions EMF gained significant effects on LTD and LTP, and on basic synaptic transmission only for exposures of excessive specific absorption rates (10 W/kg).

The already high level of Mef2 promotor activity in cells expressing PKD2 wild type is likely to be due to the fact that exogenous expression of wild-type PKD2 in C2C12 cells results in considerable activation of the kinase (data not shown).

Both RNA-Seq and qRT-PCR analyses revealed considerable activation of the ANR gene in tea but not in oil tea, which is consistent with the data from HPLC analyses of EC, EGC, C and GC contents.

As shown in Fig 3A, treatment of cells with varying concentration of Triphala for 24 h caused considerable activation (phosphorylation) of ERK without causing any change at the protein level.

These repeated findings of zero activation strongly suggest a low prevalence of false positives, while repeated findings of zero-vs.-considerable activation in the paired comparisons do suggest real group differences.

The initiating events that trigger upregulation of the corresponding genes are unclear, but there is considerable evidence for activation of PPARa, LXRa, and potentially other transcription factors that are activated by fatty acid ligands.

As discussed below, the nominal evidence for association between gout and CD14, CARD8 and IL1B, and the multiplicative interaction between CARD8 and IL1B in determining the risk of gout, support the considerable evidence that TLR-mediated activation of the inflammasome and subsequent release of active IL-1β is a central causal pathogenic pathway of gout [ 10, 32].

The linkage of improved TRAIL activity with binding and activation of CD40 has two considerable advantages compared with non-targeted TRAIL variants.

Considerable evidence indicates that prior activation of these systems modulates defensive behaviors, which facilitates somatosensory capability [51].