Am I one of those caregivers who consider anxiety to be one thing only, always requiring my favorite remedy, be it based on neurotransmitters, attachment theory, or exposure and response prevention?
Although that study did not specifically consider anxiety disorders as a severity factor, many studies have demonstrated that the course of illness is more severe in the presence of a comorbid anxiety disorder (e.g., Hawke et al. 2013; Freeman et al. 2002).
Let us consider anxiety disorders that have been associated with abnormal activity in distinct brain networks or modes including hippocampus anterior cingualte, insula, basal ganglia, and some others [63], [64].
Extending this model to consider anxiety in particular leads to several initial hypotheses.
Given the higher prevalence of anxiety primary care physicians should consider anxiety as well as depression as premorbid risk factors of dementia to improve early recognition and facilitate greater access to services.
Given the lack of research on coparenting specifically addressing anxiety, we will consider anxiety broadly, including anxiety symptoms, disorders, and child temperamental predispositions of anxiety (i.e., behavioral inhibition and negative reactivity); where empirical work on anxiety per se is quite scarce, we consider existing work on internalizing problems.
The results of the nominal group discussions are illustrated in Figure 2. Generally, the experts consider anxiety and depression to be the dimensions that most severely affect HIV/AIDS patients.
Following the established standard in child psychiatry, we consider anxiety present if reported by either the parent or the child unless there is a reason to doubt the veracity of a positive report [ 115, 116, 118, 119].
Some experts consider anxiety to be the most frequent emotional symptom exhibited by dyslexic children.[41].[41]
Indeed, none even considered anxiety a condition.
