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Autophagy is an evolutionarily conserved process that degrades cellular components to restore energy homeostasis under limited nutrient conditions.
Autophagy, an evolutionally conserved process of controlled cellular cannibalization, plays a vital role in cardiac physiology.
Our lab is focused on the molecular basis for the cellular remodeling accompanying meiosis, the highly conserved process by which gametes are produced.
These age-associated alterations in the frequency, developmental potential, and gene expression profile of human HSC are similar to those changes observed in mouse HSC, suggesting that hematopoietic aging is an evolutionarily conserved process.
Meiosis is a highly conserved process in sexually reproducing eukaryotes.
Macroautophagy (herein autophagy) is an evolutionarily conserved process, requiring the gene ATG5, by which cells degrade cytoplasmic constituents and organelles.
EMT is a highly conserved process in development, cellular physiology and wound healing in every tissue compartment.
Autophagy is an evolutionarily conserved process by which lysosomes degrade unnecessary or dysfunctional proteins and cell organelles.
Many components of this highly conserved process have been characterized and work from a number of laboratories is beginning to elucidate the mechanism by which this class of secreted growth factor triggers cellular decisions.
RNAi is a highly conserved process that protects the host from transposable elements and viruses [1], [2].
Autophagy, literally meaning "self-eating", refers to an evolutionarily conserved process for degrading organelles and cytoplasmic macromolecules.
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