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In the rd1 mouse model, retinal degeneration starts at around P11 [2] and consequently we chose this time-point for a comparative analysis of different cell death markers in wt, rd1, and PARP1 KO retina.
Consequently we chose to delete the biased item (Item 7).
It was considered relevant to explore the comments from the most distinct response groups identified in the first study and consequently we chose the extreme positive and extreme negative response groups for analyses.
Consequently, we chose to implement the simplest rule.
Consequently, we chose the DOND site (and not SEOS), which is only 40 km from PAJU.
Consequently, we chose the manual method to calculate temporal changes in crevasse area.
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Following the mass approach, we scale this feature to the population, and, consequently, we choose to change parameter (v_{{{mathrm{N}}}_{2}}) in the equation corresponding to the output of population ({{mathrm{N}}}_{2}) since this parameter represents a modulation of the threshold of the sigmoidal function (hat{mathcal{S}}).
Consequently we have chosen to report values from multiple imputation as our primary results.
Evidently, the higher sequencing coverage permits the use of larger windows covering the same physical and genetic intervals and consequently more accurate mapping, so we chose 15 SNPs as our analysis parameters.
We chose the top 0.1% candidate targets of each compound and consequently obtained 232 candidate target genes.
Still we chose this approach as pain AUC gives a time weighted and consequently a more reliable measure of pain response than single measurements.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com