Sentence examples for consequences of dysfunction from inspiring English sources

Toxicity manifest as noncancer endpoints essentially consists of the consequences of dysfunction of the whole physiological system, not of failures of any particular cell or gene.

Every year, there are over 350,000 people worldwide who are diagnosed with the tragically debilitating consequences of dysfunction in lower limb motor control, so it is obvious for the significance in searching for effective means of helping these people to regain ability to stand up and walk.

Behavioral consequences of dysfunctions of neurological origin or of aging on walking abilities can be reliably assessed by analyzing footprint patterns [6], [9], [13].

Psychiatric symptoms are most likely a consequence of dysfunction of multiple cortical areas and sub-cortical brain structures in patients with schizophrenia.

This is an important finding indicating that the function and consequence of dysfunction cannot automatically be extrapolated from an apparent orthologue.

It is presently unknown whether the intracellular protein aggregation observed in most late-onset neurodegenerative diseases is a cause or consequence of dysfunction in these pathways [ 97].

This could be at least partly explained by the alteration of sound localization and distortion leading to impaired speech intelligibility as a consequence of dysfunction of inner ear and cochlear nerve by tumor and its treatment [ 35, 36].

The reduction in nocturnal melatonin levels with the abolition of diurnal melatonin rhythmicity may be the consequence of dysfunction of noradrenergic regulation and depletion of the melatonin precursor 5-HT by increased MAO-A activity, as already seen in the earliest preclinical AD stages [ 242].

While diaphragm dysfunction might limit exercise capacity, the clinical consequences of diaphragm dysfunction in successfully liberated patients are uncertain.

Based on the pathologic consequences of metabolic dysfunction in other diseases such as cancer, we hypothesized that chronic repression of Fe-S biogenesis directly drives dysfunction of mitochondrial metabolism, cellular proliferation, and frank disease.

Whereas the nutritional consequences of CFTR dysfunction can be effectively treated by exogenous pancreas and diet, the pulmonary consequences have proved less easy to treat.