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A paradoxical type of bleeding is found when tissue materials (as the consequence of shock, trauma, burns) enter the bloodstream and cause clotting within the vessels.
The cause of death was defined for each non-survivor as related to post-cardiac arrest shock, when death occurred as a direct consequence of shock (including subsequent multi-organ failure), or related to neurological injury if this led to withdrawal of life-sustaining therapy or brain death [9].
As a major metabolic consequence of shock states, acidosis is also known to be a high predictor of in hospital mortality [ 5].
Whereas the liver plays a pivotal role in regulating a wide range of key metabolic, homeostatic, and host-defense activities, liver dysfunction is commonly viewed only as a consequence of shock and initial tissue hypoperfusion.
Because portal venous blood comprises 75% of total hepatic blood (6 ), this condition coupled with decreased hepatic arterial blood flow as a consequence of shock might have led to severe and irreversible liver damage in this patient.
The fact that prothrombin time was related to survival may reflect the importance of liver function in the pathophysiology of MALA, but may also just be a consequence of shock.
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Outcome variability can be measured, but it's difficult to know how much outcome variability is the consequence of effort, or the consequence of anticipated factors, or the consequence of shocks whose origins are publicly known and thereby insured without moral hazard.
In addition to the consequences of shock itself, the current management of hemorrhagic shock in traumatic injury relies heavily on transfusion of red blood cells (RBCs).
Herein, we illustrate the major role of NaHS in protecting the body against the consequences of shock and I/R [ 19].
Broadly speaking, adaptive capacity denotes the ability of a system to adjust, modify, or change its characteristics or actions to moderate potential damage, take advantage of opportunities, or cope with the consequences of shock or stress (24).
We hypothesised that H2S injected at the time of reperfusion could decrease the consequences of shock and reperfusion, that the use of an inhibitor of endogenous H2S production leads to opposite effects, and that adding a vascular K+ATP-channel inhibitor would improve the effects of H2S on systemic haemodynamics.
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