This was followed by 4 consecutive epidemics (minor-major-minor-major) from April 2008 through February 2010 that were all predominantly associated with group A strains.
This study was conducted to identify the prevalence of macrolide resistance among M. pneumoniae strains isolated from children with lower respiratory tract infections (LRTIs) during 4 consecutive epidemics (2000 2011) in South Korea.
Patterns of dominance in the prevalence of groups A and B have been observed to cycle in communities during consecutive epidemics (for example, A-A-B, A-A-B, etc..) and predominant genetic variants within the groups are usually replaced in successive epidemics [ 8- 10].
Using RT@HC, 95 localised epidemics were detected, 40 after epidemic declaration in the district, 20 without occurrence of an epidemic declaration in the district and 35 with consecutive epidemic declaration in the district (yielding a PPV of 63% for epidemic detection by RT@HC compared to district declaration).
To test the association of some network parameters and variables of commune infection status in three consecutive epidemic waves ("infection in 2003-4", "infection in 2004-5" and "infection in 2005-6" and "infection in any of the three waves"), several statistical tests adapted to network data were applied.
Study design: Virus isolates from infants admitted to hospital in Newcastle upon Tyne with hRSV infection during two consecutive winter epidemics were classified into lineages by genotypic analysis.
This was recently demonstrated in the "Osservatorio study", which was designed to collect epidemiological data on RSV infection in two consecutive winter epidemics (2000 2002) in Italy [ 16].
A case-control study was used to evaluate the risk factors associated with higher likelihood to acquire RSV-induced LRTI, severe enough to lead to hospital admission, over four consecutive winter epidemics in Italy (2000 2004).
Thus, 2 different MV genotypes, B3 and B2, were associated with 2 consecutive measles epidemics in Kinshasa, suggesting that MV circulation had been temporarily interrupted during the intraepidemic phase in 2004.
To determine the contribution of HBoV to New Zealand's bronchiolitis disease prevalence, we tested samples collected from infants hospitalized with community-acquired bronchiolitis (2 ) during 3 consecutive winter epidemics (June to October, 2003; July to October, 2004; and June to October, 2005) in Wellington, NZ, for HBoV by PCR.
Cocirculation of DENV-1 and DENV-2 caused outbreaks between 1996 and 1998, followed by 2 consecutive DENV-3 epidemics in 2001 2002 and 2004 2005.
