Sentence examples for conductance of action from inspiring English sources

Voltage-gated Na+ channels are essential for the generation and conductance of action potentials.

Signaling in these systems is based on fast and accurate propagation and conductance of action potentials involving voltage-gated sodium channels (Navs) (Hille, 2001; Meech and Mackie, 2007).

As the channels show outward rectification they are expected to predominantly affect neurons at the spiking threshold [15] and accordingly in our study the insulin-induced tonic conductance decreased frequency of action potential firing in the CA1 pyramidal neurons.

The action potentials were largely unaffected by the conductance increase, since fast Na/K conductances of the action potential were much larger.

In particular, no study is available for Kv7.1 channels which assemble with KCNE1 to produce the slow IKS current, a major repolarizing K+ conductance of the cardiac action potential [10].

Muscle movement is produced through excitation contraction (E C) coupling elicited by t-tubule conductance of the muscle action potential.

These results suggest that suppression of CA1 neuron intrinsic excitability, by these anesthetics, is largely due to activation of tonic GABA(A) conductances; although other sites of action may play important roles in affecting synaptic transmission, which also can produce strong neurodepression.

Hippocampal CA1 pyramidal neuron model from the paper M.Migliore, D.A Hoffman, J.C. Magee and D. Johnston (1999) Role of an A-type K+ conductance in the back-propagation of action potentials in the dendrites of hippocampal pyramidal neurons, J. Comput.

Specifically, stimulation of certain G protein coupled receptors (GPCRs) on a cell's surface leads to the opening of GIRK channels, which increases K+ conductance and opposes the initiation of action potentials.

Resting membrane potential, input conductance, current injection to threshold of action potential firing, capacitance, and membrane time constant were unaffected by time of day or the absence of BMAL1.

Addition of TTX almost completely blocked vesicular release in response to LILFU-1 highlighting the importance of Na+ conductance and action potentials in LILFU-triggered synaptic vesicle release (Figure 5F).