Sentence examples for conclusion that the function from inspiring English sources

Then Lemma 2.6 and (3.45) lead to the conclusion that the function (R_{2}(t)) is strictly decreasing if (pgeq1) and strictly increasing if (pleqsqrt{3}/2).

Then inequality (2.35) leads to the conclusion that the function (xrightarrow g p, x)) is strictly increasing on ((0, 1)) for (pin [3/2, 2]).

Inequality (2.13) leads to the conclusion that the function (phi(x)) is strictly concave on the interval ((0, 1)) and the inequality phi(x)>phi(0) 1-x +phii(1-x +phi4) holds for all (xin(0, 1)).

Then Lemma 2.3(4), (2.18), (2.20) and the monotonicity of the function (rrightarrow h_{1}(p; r)) on the interval ((0, 1)) lead to the conclusion that the function (rrightarrow h u, p; r)) is strictly increasing on ((0, 1)).

From (3.38) and (3.39) we clearly see that the sequence ({alpha _{n}/beta_{n}}_{n=1}^{infty}) is strictly increasing, then Lemma 2.6 and (3.37) lead to the conclusion that the function ((cosh t-I_{0}(t))/[cosh t-sinh t/t]) is strictly increasing on the interval ((0, infty)).

It is well known that the function (x to sin ( x )/x) is strictly decreasing on (( {0,pi /2} )), hence equation (2.9) leads to the conclusion that the function ({varphi '_{3}} ( x )/{varphi '_{4}} ( x )) is strictly increasing on (( {0,pi /4} )).

This typically leads to the conclusion that the functions of these proteins, in this case the epigenetic functions, are indeed essential for viral-induced transformation.

Other Na+/K+ ATPase inhibitors that are structurally distinct from cardiac glycosides also exhibit antimigratory activity, corroborating the conclusion that the antiport function of Na+/K+ ATPase and not the receptor function is important for supporting the motility of MDA-MB-231 breast cancer cells.

This leads to the paradoxical conclusion that, while the function of many or even most genes is buffered by a redundant paralog, redundancy is a relatively rare outcome of gene duplication.

The present findings, however, fully support the conclusion that the principal function of CXCR7 is to act as scavenger for CXCL12.

The low level of identity and similarity between substrate binding amino acids and the absence of a cholesterol catabolism phenotype and cholesterol oxidation catalytic activity lead to the conclusion that the gene function has been misassigned.