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To conclude, expression of RAS components and their cellular localization suggest their involvement in controlling aqueous humour dynamics and intraocular pressure.
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We conclude that expression of spinal G (αi, αo, αs, αq and β) protein may be up-regulated after chronic morphine treatment which could be attenuated by knockdown of spinal mGlu5 receptor with antisense ODN.
We conclude that expression of nuclear T7 constructs is not feasible in tobacco cells, but cytosolic transcription provides an alternative means to over-express RNAs directly in the cytosol.
We conclude that expression of ezrin in hematopoietic-lineage cells is not required for lymphoid development.
We conclude that expression of EEF1A2 is progressively upregulated during the progression of normal plasma cells to MGUS and MGUS to MM.
We conclude that expression of A-type lamins in CRC promotes tumour invasiveness through reorganisation of the actin cytoskeleton.
Therefore, it is reasonable to conclude that expression of the endogenous lipid motif on P6 and recognition of this motif by TLR2 is required for effective immunization.
From this, we conclude that expression of Lrp5 protein is restricted to cells in the basal lineage based on a post-transcriptional mechanism.
We conclude that expression of NTPD8 may be low and that antibody staining is not sufficiently sensitive to define its location with certainty.
From these data it is not formally possible to conclude that expression of the Core MYC signature orchestrates the typically aggressive behavior of the basal-like breast tumors; however, it is likely that this expression profile is not inconsequential.
We conclude that expression of NESP55 is a sensitive marker for hypoxia-dependent chromaffin metaplasia in neuroblastoma, which represents the dominant form of differentiation in this group of tumors.
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