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Proof of concept experiments were performed to demonstrate the viability of the system for sequencing.
To demonstrate this concept, experiments are performed with a 9.4-cm-long, 1.6-cm-diameter LN crystal mounted within a vacuum chamber while monitoring the crystal end velocity.
Proof of concept experiments have been successful illustrating that minichromosome platforms can be modified in vivo.
Proof of concept experiments have shown that tissue factor/factor VIIa inhibitors have antithrombotic activity without enhancing bleeding propensity.
These proof of concept experiments give rise to possibilities to translate the transduction of neurofibromin to in vivo experiments with the goal of a restoring of lacking protein in all body cells.
These proof of concept experiments had established LBTs as useful components of LRET assays, but they had not yet been combined with fluorescent proteins as acceptors to investigate PPInts of full-length proteins or individual domains.
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These proof-of-concept experiments were performed with a high cell dose of 4 × 106 iPSC-Mac per single intra-pulmonary application.
Finally, we utilize proof-of-concept experiments to suggest that our analyses can quantify the behavioral effect of neuropharmacological agents and neural perturbations with optogenetics.
The paper demonstrates this operating capability with proof-of-concept experiments using The University of Queensland's X3 impulse facility.
In our proof-of-concept experiments, we deposited conformal FP layers onto the surface of flexible carbon nanotubes (CNT).
In proof-of-concept experiments, the group showed that by shining the light on groups of neurons thought to be involved in pain and modified to be light-sensitive, they could induce behaviors associated with pain.
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