Therefore, it is suggested that when the stress concentrations are caused, a liquid phase is needed in order to relax the stress concentrations, however, when no stress concentrations are caused, a liquid phase is not needed for superplasticity.
This again suggests that the observed current inhibition at high substrate concentrations is caused by internal substrate accumulation.
On the other hand, in alcoholic liver fibrosis it has been shown that elevated PIIINP concentrations are caused by increased histologically confirmed fibrogenesis [ 21].
It is rather indicated that low plasma carnitine concentrations are caused by a low availability of precursors for carnitine synthesis during gestation.
The authors have concluded that the early rise in circulating β-carotene concentrations is caused by the intestinal input, whereas hepatic secretion is the source of later increases.
While loading the tibial tray, high stress concentrations are caused by the stem; the cancellous bone can collapse, leaving a void around the stem.
Hence, we suggested that low plasma carnitine concentrations are caused by a reduced carnitine synthesis due to a reduced activity of iron-dependent enzymes involved in carnitine synthesis.
In patients traditionally categorized as having "pre-renal" AKI, temporary increases in serum creatinine (SCr) concentrations are caused by changes in fluid status and/or renal perfusion.
Rather, a first possible scenario is that the reduction of the choice and feeding scores for high sugar concentrations is caused by an inhibition from, for example, high-osmolarity sensors specifically upon the reflexive pathways.
More likely, changes in Hb concentration were caused by fluctuations in plasma volume by other mechanisms, as has been reported in the time course of surface cooling [ 48, 49] and rewarming [ 50].
