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Amplification of RT-cDNA was initially performed on a control specimen at different concentrations to define a linear range for all genes.
Amplification of RT-cDNA was first performed on a control specimen at different concentrations to define a linear range for all genes.
Furthermore, the authors used mean values of blood creatinine concentrations to define the study outcome, without using a specific cut-off that is needed to identify cases of acute kidney injury.
Moreover, for quantitative measurements, that is, determination of metal ion concentrations within cells, it is essential to perform measurements at a range of concentrations to define whether the resulting measurements are influenced by the sensor concentration.
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While the blood glucose concentrations used to define diabetes and impaired fasting glucose are somewhat arbitrary, they do correlate with the risk of macrovascular and microvascular disease.
Hemorrheologic abnormalities in familial hypertriglyceridemia (low atherosclerotic risk) were at least as marked as in dyslipoproteinemias with high atherosclerotic risk, suggesting that it might be most important to determine lipoprotein concentrations and to define exactly the type of dyslipoproteinemia for estimating the individual cardiovascular risk in these patients.
"Carbon" is not carbon dioxide, a natural substance not toxic to humans at many times current ambient concentrations; and to define carbon dioxide as "pollution" is an attempt simply to assume the answer to the central policy question.
Community pollution concentrations help to define the background or baseline conditions for particular media and for a particular segment in the NCS location.
The two methods have been applied to a PK study in subjects treated with diosmin (450 mg per os), in order to measure diosmetin concentrations and to define the most relevant type of diosmetin conjugates present in body fluids.
Our inability to detect a lower-affinity binding site may reflect the technical difficulties involved upon examination of binding at the high radioligand concentrations needed to define the low-affinity binding site.
Additionally, the critical concentrations used to define qualitative resistance for second-line medications remain a subject of debate, are influenced by the platform of testing, and ultimately require rigorous clinical follow-up among large numbers of patients [ 33].
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