C7a also induced an increase in cytosolic calcium concentration, mainly from intracellular compartments, and a significant decrease in the ATP levels.
Model projections suggested decreases (~ 10 60%) in pollutant concentrations, mainly attributable to decreases in pollutants from local sources between 2001 and 2010.
Elevated Ni and Hg concentrations resulted mainly from the nature of the parent rock material.
This rise in the cytosolic calcium concentration comes mainly from the intracellular calcium stores, since it was not dependent on the extracellular calcium concentration.
The large difference in concentration distributions stems mainly from the sizeable number of undetectable levels in the measured samples.
In fact, the large difference in concentration distributions stems mainly from the large number of undetectable levels.
It is concluded from these results that the α-tocopherol concentration increase resulted mainly from the increase in α-tocopherol content.
In urban areas, high concentrations of PM derive mainly from motor vehicle emissions.
Genetic links to serum urate concentrations have been identified, mainly from earlier studies of monogenic disorders, but have also been recently analyzed using genome-wide association approaches.
Previous studies have suggested that the reduction in plasma HDL-cholesterol concentrations with TFA intake (mainly from PHVO) compared with cis-MUFAs may be imputed to increased CETP activities.
