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A study of female human exposure levels of PFOS concentration found that levels in the United States are approximately twice those in Japan (Olsen et al. 2003b; Taniyasu et al. 2003).
In Bangladesh, a study of the pregnancy outcomes of 2006 women who regularly consumed drinking water with high arsenic concentration found that there was a small but statistically significant association (OR: 1.005, 95% CI: 1.000-1.010) between arsenic exposure and congenital anomalies, though the study did not specify the nature of these anomalies [ 24].
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A study in Bangalore, South India, where mothers had higher vitamin B12 concentrations, found that low maternal vitamin B12 concentrations predicted fetal growth retardation [ 30].
Studies that have looked at both urinary and toenail As concentrations found that the relationship between these biomarkers and drinking water As concentrations are nonlinear at low drinking water As concentrations but become linear as drinking water As concentrations increase (Karagas et al. 2000; Kile et al. 2005; Watanabe 2001).
Despite its cytotoxicity at relative high concentration, we found that low doses of CPT exhibited overt hormetic and neuroprotective effects in PC12 cells.
We found that low concentrations of thrombin (1 nM) enhances neurite growth and branching, neuron viability, and protects against excitotoxic damage.
We found that low concentrations of MegaFasL sensitised GIST cells to imatinib-induced apoptosis.
Finally, in our analysis of AChE concentrations, we find that exposure to sublethal concentrations has a lasting legacy on tadpole AChE concentrations.
First, for each concentration of antibiotic, we find that growth is maximal at a single salicylate concentration S*.
Prior concentration studies found that 75 µM concentration provided optimized sensograms for all proteins.
We further optimized the IPTG concentration and found that the most suitable IPTG concentration for inducing GST-CTCF-ZFs expression was 0.6 0.7 mmol/L (Fig. S1C).
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