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We speculate that a spontaneously high serum IgG concentration at birth reflects placenta function and is an indicator of a more mature immune system capable of protecting the preterm infant against severe neonatal infections.
Haemoglobin concentration at birth c.
Infants are also likely to have a higher manganese concentration at birth.
This is consistent with our finding of a correlation between plasma/blood Pb ratios and cord blood Pb concentration at birth.
Mean serum 25(OH D concentration at birth for the EPTI cohort was 46.3 nmol/L (SD 14.0) with lower concentrations in infants born <28 weeks PMA (42.0±9.8 nmol/L) than at 28 32 weeks (51.8±19.5 nmol/L), p=0.02.
We used the model output of lipid-normalized human contaminant concentration at birth (nanograms per gram lipid) to assess prenatal exposure, and postnatal and childhood exposures were defined as the average concentrations encountered from 0 6 months (the duration of breastfeeding) and 0 9 years, respectively.
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Conclusions: Prophylactic immunotherapy with IVIgG did not improve the immune competence in preterm infants with low serum IgG concentrations at birth.
Mercury exposures prior to 3 months preconception are assumed to have a negligible contribution to blood mercury concentrations at birth.
Additional control for maternal factors did not materially alter the association between predicted serum PFOA concentrations at birth and eGFR.
The significantly higher maternal serum concentrations of these PFASs at 16 weeks than both the maternal and infant's concentrations at birth suggest transplacental transfer of PFASs.
More than 90% of women had detectable urinary BPA concentrations at 16 and 26 weeks of gestation, and 87.1% had detectable concentrations at birth.
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