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The barley mutant nec1 comprising of a frame-shift mutation of CNGC4 displays a necrotic phenotype and constitutively over-expresses PR-1, yet it is not known what effect the nec1 mutation has on barley resistance against different types of pathogens.
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Thus we examined the possibility of a frame-shift involving the preceding ORF pheT, coded in a different frame, as a potential mechanism to explain this observation.
‡ Candidacy is based on the presence of a frame-shift and the length of the repeat tract.
It should be noted that SSH629-D8 appears to be part of a pseudogene containing a frame-shift mutation.
Retention of the fourth intron caused a frame-shift mutation and changed subsequent amino acids.
This deletion of 11 bp leads to a frame-shift and premature Stop codon.
In the present report, we characterized a frame-shift mutant of α1S that expresses two complementary fragments of α1S.
The deletion of exon 22 in Kcnt2 causes a frame-shift and the predicted residual protein is Slo2.1(1 828).
This results in a frame-shift and a premature termination of translation.
The coding sequence of FAD3-2.2 contains multiple in-frame stop codons and a frame-shift mutation and thus is likely a pseudogene.
The deletion of two nucleotides within human CTI exon 1 would produce a frame-shift (as depicted by the +1 and +2 reading frames).
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