Sentence examples for comprehension of pathophysiology from inspiring English sources

Developments in the monitoring of the microcirculation illustrate how sophisticated technological tools may improve our comprehension of pathophysiology, as well as their limits to implementation into routine clinical practice.

Thus, special journal issues providing visibility to new upcoming idea and hypothesis in resuscitation basic science, translational studies, and bioengineering, like the present issue, have to be supported, paving the way towards a better comprehension of pathophysiology, mechanisms, and management of sudden cardiac arrest and amelioration of resuscitation care.

The emerging fields of functional genomics and functional proteomics offer the opportunity to translate these advances into a full comprehension of the pathophysiology of cancer.

Sepsis remains an important cause of mortality in intensive care unit (ICU) despite a better comprehension of its pathophysiology [1, 2].

The current evidence in using metabolomics in atherosclerotic cardiovascular disease is also limited, and more well-designed studies remain to be established, which might significantly improve the comprehension of atherosclerosis pathophysiology and consequently management.

These examples not only emphasize how translational research resulted in major improvements in the comprehension of the pathophysiology of severe clinical conditions and offered promising perspectives in critical care medicine but also point out the obstacles to translate such achievements into clinical practice.

Experimental models of diabetic retinopathy (DR) have had a crucial role in the comprehension of the pathophysiology of the disease and the identification of new therapeutic strategies.

The main goal of these human studies is a better comprehension of the pathophysiology of such disorders as well as identification of biomarker candidates and potential novel targets.

A mixture of diverse animal models of FMS, each mimicking particular biomarkers and clinical conditions observed in FMS, may contribute to the comprehension of its pathophysiology and improvement of its management.

Advanced human-specific cell- and tissue-based models (e.g., Singh et al. 2011) and next-generation tools are making possible a fuller, dynamic comprehension of disease pathophysiology and a more reliable and cost-effective drug discovery process (Muotri 2015).

Understanding how these more newly identified GLUT isoforms contribute to regulating myocardial glucose transport will enhance our comprehension of the normal physiology and pathophysiology of the heart.