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It has long been recognized that aphasic patients display sentence comprehension deficits.
Patients with naming impairments in the context of relatively mild comprehension deficits are most likely to benefit from this approach, while avoiding the negative consequences of over-generalisation.
Speculatively, one may argue that comprehension deficits resulting from lesions within the inferior frontal cortex, as well as production deficits resulting from lesions within the superior temporal cortex, may arise from disruption of lower-level information processing.
Word-finding difficulties and comprehension deficits despite relatively preserved cognitive functions are characteristic symptoms during the first two years, and distinguish PPA from other dementia types like Alzheimer's disease.
The majority of patients with comprehension deficits failed to perceive them.
They can include, but are not limited to, speech production and speech comprehension deficits.
Group interaction obviously clarified some misunderstanding and comprehension deficits since the mean average of subjective comprehension after discussion increased slightly to 5.82.
In later stages, patients have auditory comprehension deficits for even one-step commands, and exhibit alexia, and agraphia [1], [4].
However, our study additionally suggests that visual word comprehension deficits in reading disability may be due in part to a deficit in the direct mapping from orthography to semantics.
Comprehension deficits are common in both Wernicke's aphasia and semantic aphasia.
Patients with severe comprehension deficits who were not able to fully engage in therapy were excluded.
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