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The SAG compound was then tested for anti-inflammatory properties relative to SA and acetylsalicylate (aspirin).
The compound was then transplanted autologously into bone defects of rabbits to observe the vascularization in vivo.
The ability of the synthetic compounds for extraction and phase transfer of glucosamine, as a hydrophilic organic compound, was then studied.
In the first step, Ni-TPP is reversibly hydrogenated to nickel 5,10,15,20-tetraphenylchlorin (Ni-TPC); this compound was then hydrogenated to nickel 5,10,15,20-tetraphenylisobacteriochlorin (Ni-TPiB).
This compound was then characterized by X-ray crystallography (Figs. 2, 3) [37].
The in vivo inhibition rate of each compound was then calculated according to the following formula: Inhibition rate % = av local lesion no. of control − av local lesion no. of drug-treated / av local lesion no. of control × 100%%.
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The unique chemical signals for each trace compound are then compiled into a database.
The prediction of Vss for a new compound is then performed in two stages.
Potential molecular formulas of the compound are then sorted with respect to this score.
The sodium compound is then is heated with water and the olive oil in large copper vats over fermentation pits.
A curium compound is then selectively extracted using multi-step chromatographic and centrifugation techniques with an appropriate reagent.
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