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The latter compound was subsequently deprotonated and used to polymerize ethylene oxide by a classical core-first approach.
The purified compound was subsequently subjected to 1H and 13C NMR spectroscopic analysis.
Each compound was subsequently associated with a set of genes deleted from strains that exhibited significant fitness defects induced by the compound.
However, a phase 2 trial demonstrated that only 19.7% of patients with mild-to-moderate AD developed a predetermined antibody response [ 15], and this compound was subsequently discontinued because meningoencephalitis was observed in 6% of patients [ 16].
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Then, Al element and Al2OC compound are subsequently produced with the reduction in the release of gas CO as well with continuous environmental heating.
This compound is subsequently converted into 3,4-dihydroxyphenylacetic acid (DOPAC) by aldehyde dehydrogenase.
The formazan compound is subsequently solubilized by a solubilization solution and the absorbance of soluble formazan can be measured spectrophotometrically [ 82].
After 12 h, 10 μmol/L of compounds was subsequently added and incubated for 48 h.
The potency of the isolated compounds was subsequently investigated by in vitro kinase assays.
Five compounds were subsequently selected for experimental assay.
These bioactive compounds were subsequently tested in a tertiary polyglutamine aggregation assay, which identified five inhibitors.
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