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In this study we show that CTBT, a compound enhancing the antifungal activity of several drugs [ 2], generates superoxide and other reactive oxygen species (ROS) and induces massive oxidative stress in yeast cells which enhances the antifungal activity of several unrelated drugs.
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The compound enhances its potency in vitro and reduces cell toxicity in vivo.
In addition to this, the presence of IL compound enhances the capacity density of battery reaching to the maximum value (2554 mAhg-1) at 1.5 mM of IL.
A pure pheromone stimulus following a mixed compound/pheromone stimulus was generally not affected, with two exceptions: one compound enhanced and another inhibited a subsequent stimulus.
Cheng et al. [53] reported that this compound enhanced the efficacy of cardiogenic differentiation of endogenous bone marrow derived from mesenchymal stem cells.
Importantly, this compound enhances neuroregenerative effects; therefore, it shortens the required duration of immunosuppression [ 4– 6].
However, this compound enhanced the effects of the siRNAs on recovery of E-cadherin expression, especially in Hs578T cells (Fig. 1B and C).
And like our studies, other group suggested that melittin, a bee venom toxin compound enhanced TRAIL-induced apoptosis by activating JNK/p38 pathway [ 51].
These results confirm the systemic immune-regulatory effects of YCW and reveal that this compound enhances cell-mediated immune responses in young animals.
The structure of the phenolic hydroxyls esterized of the compound enhances the stability and liposolubility as well as easily hydrolysed and readily release bioactive [ 10].
Antimicrobial combinations are extensively tested in vitro in the hope of obtaining a synergistic interaction [ 25], that is, a phenomenon in which one compound enhances the individual activity of another compound in combination and vice versa [ 26].
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CEO of Professional Science Editing for Scientists @ prosciediting.com