Sentence examples for completion deficit from inspiring English sources

This pattern completion deficit during recall may lead to a false interpretation that the NMDA receptor in the hippocampus is required for associative memory recall [8].

Our both pharmacological and inducible genetic experiments suggest that such pattern completion deficit, if any, is unlikely due to the direct effect of the NMDA receptor on memory recall.

Of course, it might be equally possible that the originally reported minor pattern completion deficit in CA3-NR1 knockout mice may have resulted from behavioral variations during those particular sets of experiments (it is noted that an unusually large group of control mice were used in comparison with a much smaller group of mutant mice).

Lack of apparent requirement of CA1 NMDA receptors for the pattern completion has led us to entertain the following two alternative scenarios: 1) CA1 might be just one of many regions involved in retrieving memory, the knockout of CA1 NMDA receptor is insufficient to see the pattern completion deficit.

Thus, these experiments suggest that pattern completion deficits originally observed in the DAT+/− mice may be caused by the dopamine imbalance.

It will be important in future studies to further define the anatomical loci from which the observed pattern completion deficits originate.

Thus, memory pattern completion deficits observed in the DAT heterozygous mutant mice may be due to the mouse's inability to meet the increased attentional demands during partial cue-based memory recall as a result of synaptic dopamine disturbance.

Eli Manning had one completion and a 17-point deficit at that point, and soon fumbled away the last reasonable chance at a comeback.

The 57-yard completion erased a 4-point deficit to the San Diego Chargers with a little more than two minutes left and rallied the Packers to a 31-24 victory.

While the dopamine system is well known to be crucial for the regulation of many cognitive processes [8], [16] [18], [23] [25], our present study provides evidence for the first time that dopamine imbalance, resulting from the loss of one allele of the normal dopamine transporter gene, caused a specific deficit in pattern completion during associative spatial memory recall.

Although a proportion of patients may recover this deficit spontaneously after completion of therapy, some individuals potentially struggle to return to their preillness level (Koelwyn et al, 2012), emphasising the need for countermeasures protecting against therapy-related physiological deterioration.