Sentence examples for competing secondary from inspiring English sources

We simulated co-transcriptional RNA folding dynamics to identify competing secondary structures that might interfere with terminator folding kinetics or impact termination activity.

These mutations, which were predicted to alter the relative stability of competing secondary structures within the precursor, changed the distribution of alternative RNA structures that are resolved in native-gel electrophoresis.

The proposed probabilistic and non-greedy access mechanism allows competing secondary flows to efficiently and fairly share the available spectral opportunities without relying on the common control channel for inter-flow communication.

Displacement of single stranded 3'-ends would be energetically favorable through a branch migration mechanism [ 13, 14] with a resulting equilibrium between competing secondary structures.

Furthermore, active secondary metabolic gene clusters of such strains may be silent to prevent diversion of precursors into competing secondary metabolic pathways, thus, facilitating enhanced production of the desired compounds [ 38].

If not, it would imply the downstream behavioral response should be both labile and very finely graded, i.e., subject to numerous competing secondary modulations of equal strength, which may or may not correlate with what the behavioral data is showing.

Considering the performance of existing competing technologies, secondary prevention was ranked lowest.

Although such competing RNA secondary structures have not yet been found in all clusters of MXEs [ 30, 31], their identification would provide strong further confidence to any prediction.

These experiments highlight the importance of competing, alternative secondary structures in the nascent RNA and their folding dynamics, as a basic mechanism through which small molecule effectors and transcription factors can control gene expression.

In the subgroup of patients with CV disease history at baseline (n = 336), RHR was found to be associated with the incidence of primary outcome (P = 0.0002) but also with renal risk alone, adjusted for all-cause death as a competing event (secondary outcome; P < 0.0001).

In some of the best analysed genes, the Drosophila DSCAM (Down syndrome cell adhesion molecule), Mhc (muscle myosin heavy chain) and 14-3-3-ζ 14-3-3-ζ 14-3-3-ζ 14-3-3-ζenes like dynamin-1[ 2], splicingenesthe MXEs is regulated butcompeting RNalsocondary structures formed by conserved motins withumanhe introns and complementary acceptor sequences in either the 5' or 3' intron [ 3, 4].