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Correlation between gene silencing and localisation to transcriptionally repressive heterochromatic compartments has been reported in mouse cycling lymphocytes [55], [56], [57], human and mouse erythroid cells [58], [59], [60] and retinoblastoma cells [61].
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Many of the Rab proteins that are localized on distinct compartments have been reported to coordinate sequential steps of membrane transport [20], [21].
Changes in various cell activities and in the structural organization of subcellular compartments have been reported as accompanying the microspore reprogramming process in some herbaceous and woody species [ 4- 11].
Thus, while a reduced memory B compartment has been reported in cancer patients, we show that a melanoma-reactive portion of this compartment remains in our patient cohort.
Differential Sca-1 expression in the luminal compartment has been reported to delineate populations enriched in hormone receptor-positive and hormone receptor-negative cells, both containing clonogenic progenitors (Sleeman et al., 2007; Regan et al., 2012).
Given that the blood DC compartment has been reported to include different DC lineages including myeloid (CD11c+DC) and plasmacytoid (CD123+DC) DC (Robinson et al, 1999), we assessed the DC subset distribution in these patients.
Several groups have reported procedures for biochemical enrichment of bacteria-containing vacuoles (BCV) but no proteomic analysis of such a compartment has been reported (Kovářová et al., 2002 analysed Francisella tularensis-containing phagosomes by 2DGE but only reported the identities of two host-derived proteins).
Genes acting at the extracellular compartment have been reported to exhibit high rates of evolution [ 20, 47].
Alterations in the peripheral B cell compartment have been reported in T1D patients; however, to date, such studies have produced conflicting results and have been limited by sample size.
Multiple alterations in the composition of the B-cell compartment have been reported in SLE, arguably the autoimmune disease with most florid and variable changes in B-cell homeostasis.
This becomes apparent upon BFA treatment because of TGN aggregation into BFA compartments, as has been reported earlier for HS::secGFP (Viotti et al., 2010).
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CEO of Professional Science Editing for Scientists @ prosciediting.com