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This compartmentalization may be true for RGCs.
Such a compartmentalization may be due to a differential distribution of the target mechanism or the target structure of NO, Cx37.
These results suggest that nuclear compartmentalization may be associated with post-natal GKRP co-expression, a condition that has not been analysed in hypothalamic tanycytes or liver cells.
The hypothesis of GR compartmentalization may be supported by the fact that LPS and IL-1β induced GR upregulation without increasing GR mRNA [ 69].
Although the significance of nuclear CCR3 localization has not yet been investigated, the nuclear compartmentalization may be the result of a transcription-factor-like function, involved with maintenance and growth under normal conditions.
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The different compartmentalization patterns may be explained by differences in host genetic susceptibility, anatomical features, the genome of the infecting viruses, as well as high rates of mutation and replication, generating a quasispecies [ 37], and variants which may escape from surveillance [ 38].
This may simply reflects the fact that compartmentalization may exist within different rice prolamins, but could not be detected given the broad spectrum nature of the prolamin antibody.
In summary, our data demonstrate that epidermal stem cells are lineage restricted during homeostasis and suggest that compartmentalization may constitute a conserved mechanism underlying epithelial tissue maintenance.
In recent years evidence implied that this compartmentalization may not be restricted to cytoplasm and mitochondrion only, but may also extend to the cellular level.
It has been suggested recently that the degree and compartmentalization of ATP depletion may be defect specific and may thus have also specific biological consequences [ 27].
The neural localization of the 50 kDa species and its nuclear compartmentalization suggests that these may be key Thap1 species controlling neuronal gene transcription.
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