Sentence examples for common complements from inspiring English sources

We also explain how this result is linked to the problem of finding common complements for two closed subspaces of H.

However common complement escape principles are, emerging in terms of conserved binding repertoire for host regulators and evasion among the large variety of infectious microbes.

In this paper we find a necessary and sufficient condition for two closed subspaces, X and Y, of a Hilbert space H to have a common complement, i.e. a subspace Z having trivial intersection with X and Y and such that H=X+Z=Y+Z.

If E and F are tests and E⊆F, then we have F~E (since the empty set is a common complement of F and E); since E⊥(F / E), we have F⊥(F / E) as well, and so (F / E) is empty, and F = E.

Previously, association mapping was used mainly in human genetics, but it is now becoming a common complement to biparental crosses in the genetic mapping of quantitative traits in plants [16], [17], [18].

This is because of common causal factors, such as common complementing treatment methods, common background mortality and so on.

The common complement pathway results in formation of the terminal complement complex, that is, the membrane attack complex (MAC, C5bC6C7C8C9).

C3 activation is upstream to several other steps in the common complement pathway, such as activation of C5, release of C5a, and formation of C5b-9.

For example, greater variability across regions suggests that each region has a unique complement of trans-acting proteins guiding this process that is stable across individuals, while greater variability across individuals suggests that each region contains a common complement of trans-acting factors that can vary across individuals, among possible interpretations.

Therefore, the finding that mice depleted from circulating C3 by cobra venom factor as well as mice deficient in the common complement pathway component C5, its receptor C5aR, or the alter-native pathway component factor B were completely protected against anti-MPO-induced glomerulonephritis was unexpected [ 29, 30].

These pathways, which proceed with sequential activations of proteases, converge at the formation of C3 convertase (C3bBb) which acts on intact C3 to generate more C3b and forms C5 convertase (C3bBbC3b) which acts on intact C5 to continue in the common complement pathway.