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Viewed from this perspective, our culture appears to be a multifaceted feedback mechanism engaged in the process of satisfying, reinforcing, struggling with, and manipulating our cognitive predispositions.
The cultural manipulation of our cognitive predispositions is the topic of the present essay, which discusses several examples of such manipulation that respond to our desire to assign a certain "truth-value" to a given narrative, i.e., to attach a label to it announcing its "factuality" or "fictionality". "Truth-value" is a loaded term, resisting definition, and liable to lead me into trouble.
22 An inherent weakness of these debiasing approaches is that they target the same human tendencies to think and problem solve in a certain way (also termed cognitive predispositions to respond) that often results in the correct diagnosis.
Neuropsychological studies have proposed that an increased emotional awareness (Novick-Kline et al. 2005), intolerance of uncertainty (Dugas et al. 1998), and a cognitive style termed looming vulnerability (Williams et al. 2005), i.e., mental representations of dynamically intensifying danger, appear to be among the cognitive predispositions to generalized anxiety disorder.
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In her new book, she gives herself a refreshingly modest assignment: to demonstrate that a certain cognitive predisposition has contributed to the development of, and continued interest in, specific literary motifs that occur across a wide variety of cultures.
What matters for Zunshine's argument is simply that humans have a cognitive predisposition to divide the world into two categories of things: objects, which have functions, and beings, which have essences.
It is this cognitive predisposition that enables the power structure to thrive, and consequentially, why we allow evil to flourish.
RASopathies, a family of disorders characterized by cardiac defects, defective growth, facial dysmorphism, variable cognitive deficits and predisposition to certain malignancies, are caused by constitutional dysregulation of RAS signalling predominantly through the RAF/MEK/ERK (MAPK) cascade.
Behavioral therapy and nutrition education might be sufficient to engage cognitive control and counteract predispositions in this population, which, if our findings are replicated, would be encouraging.
Their effects range from vulnerability to stress in later life to cognitive performance, to emotional predispositions, to enduring changes in neurotransmitter function and brain morphology.
Recent data suggest a genetic predisposition for cognitive decline after cardiac surgery in patients possessing the apolipoprotein E epsilon-4 allele, known to be associated with late-onset and sporadic forms of Alzheimer's disease.
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