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Transplant-induced HO-1 protein expression was downregulated in CO treated recipients at 6 and 24 hrs post-reperfusion.
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Furthermore, cytochrome c, a marker for cellular injury, was lower in the isoflurane and DTX co-treated cells when compared with DTX alone, isoflurane-propofol DTX co-treated and isoflurane-propofol DTXTX groups.
To measure NF-kB activation, monolayers were either pretreated (1 h) with melatonin, and then co-treated with TNF-a (30 min), or treated without test agents and co-treated with TNF-a (30 min), or not treated (controls).
Significant inhibition of HAV was observed only when HAV was co-treated with extracts.
Wild-type NSCs were co-treated with H2O2 and cycloheximide.
Included are 526 children, 294 (56%) co-treated for TB.
Cells were co-treated with 10 µM deprenyl or 500 nM apocynin.
To further test this possibility we co-treated curcumin-stimulated cells with the PTPC inhibitor, cyclosporin A (CsA) [26].
The stimulation of IL-18 processing by LeTx was reduced in cells that were co-treated with celastrol (Fig. 5).
Both cells were co-treated with 10 μM aPPD.
This protective effect was also observed in antimycin A and MB co-treated cells.
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