Exact(3)
At a median follow-up of 28 months, there were no carcinoma recurrences in the CNS; 1 patient had a recurrence outside the CNS.
As a rule, certain morphological features aid in the distinction between secondary and primary neoplasms of the CNS (1).
The exact role that PrPSc plays in prion disease remains unclear, but PrPSc is known to accumulate in the central nervous system (CNS) (1 ).
Similar(57)
It is upregulated in inflammatory-active brain lesions of patients with multiple sclerosis (MS), a chronic degenerative autoimmune disease of the CNS [ 1, 16, 45].
Vertebrate achaete-scute and atonal genes probably also have proneural functions but in a much more limited set of cells, in particular in the CNS [ 1, 2, 4].
Multiple sclerosis (MS) is a chronic autoimmune disease that affects the myelinated axons in the central nervous system (CNS) [1].
Primarily dopaminergic neurons synthesize dopamine in the CNS [1], while neuronal fibers, adrenal medulla, and neuroendocrine cells synthesize dopamine in the peripheral nervous system [1].
Oligodendrocytes (OLs) are the glial cells that produce myelin in the central nervous system (CNS) [1], [2].
This stage is followed by the meningoencephalitic stage 2 during which the parasites invade the central nervous system (CNS) [1].
Multiple sclerosis (MS) is considered a paradigmatic autoimmune inflammatory disorder of the central nervous system (CNS) [1], [2].
Cerebral cavernous malformations (CCM) are congenital or sporadic vascular disorders of the central nervous system (CNS) [1] [17].
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