Sentence examples for cms trait from inspiring English sources

Both the nap and pol CMS trait was selected during the course of conventional breeding, apparently because in a particular confluence of nuclear genotype and environment, the copy number of the critical sublimon amplified sufficiently to exert a phenotypic effect (Figure S3).

It was also found that expression of orf224/atp6 chimeric gene is correlated with CMS trait in Brassica napus[ 51].

The mitochondrial gene orfH79 is a candidate gene for causing the CMS trait in CMS-Honglian (CMS-HL) rice.

In some cases, these differences helped to identify genetic elements responsible for the CMS trait [ 18- 22].

The other six sources have not been able to be used commercially, because they express the CMS trait at an adequate level.

Since ORFH79 is present mainly in mitochondria of YtA, and importing ORFH79 into mitochondria can induce the CMS trait, which inspired us to focus on the mitochondrial activity.

For adjusted GR83-98, a similar situation occurred on OAR4 (24 cM and 28 cM), and on OAR22 for the same trait (68 cM: -1.75 SD, and 88 cM: +1.90 SD) with the QTL being mapped to separate marker bracket intervals.

Furthermore, on chromosome 5H at 45 cM significant marker trait associations for BY, SPAD and OA were detected in the stress treatment, and at 95 cM significant associations for BY and SPAD in the control treatment.

A QTL segregating at 44 cM affecting the trait was simulated while assuming a paternal effect of the IGF2 locus.

An additive effect of gene action appeared in the D trait (20.26 cm in CC, 21.43 cm in CT and 22.31 cm in TT).

Splenic reduction (median [range]) was observed in women with and without abnormal hemoglobin-typing (available for 30 women): 4 (0–8) cm with normal hemoglobin typing (N = 16), 2.5 (1–6) cm with homozygote β-thalassemia (N = 4); 3 (1–7) cm with β-thalassemia trait, N = 9; and no change in one with hemoglobin-E disease (P = 0.381).