Sentence examples for cms in human from inspiring English sources

Both intrinsic and extrinsic pathways involve activation of apoptosis by CMs in human cancer cells.

The need for these high level structures arises from a packaging problem: the DNA molecule is long (chromosomes range from 1.7 to 8.5 cm in human) and negatively charged, resulting in electrostatic repulsions that stiffen the polymer and prevent it from fitting inside the nucleus.

LOR d) was computed for all Pfam domains in CM and non-CM genes in human.

Acetic acid injection and ethanol injection are considered to be a rational alternative to surgical resection and, in particular, PEI seems to be the safest and most effective percutaneous treatment for resectable and unresectable HCCs <3 cm in humans [ 6, 12- 14].

Indeed, owing to the paucity of genes annotated with CM function in human, the negative reference set (non-CM proteins) used for both training and testing is most likely 'contaminated' with CM proteins yet to be discovered.

The reason why we focused on the difference between these two groups was because we aimed to find the genetic basis of the differences in height between Pygmy and non-Pygmy populations, instead of simulating the human height itself (i.e. the basal 140 cm in all human populations).

This chapter reviews the acute behavioral effects of organophosphates (OP) and carbamate (CM) pesticides in humans and animals.

Data on the duration and significance of renal vasoconstriction and hypoxia after CM exposure in humans remain controversial [ 4].

Curley et al (1995) are reporting on the induction of a significant tumour necrosis and reduction in tumour volume in lesions as large as 12 cm in diameter in human liver malignancies.

The hypothalamus is a subcortical region with roughly 4 cm neuronal tissue in human brain [ 15] located at the third ventricle, rostroventral to the thalamus.

While it is not possible to identify the cellular source of newly forming CMs in the adult human heart, several studies in mice have addressed whether CM renewal during homeostasis and the low-rate CM replacement after cardiac injury are due to proliferation of existing CMs or due to CM differentiation from progenitor cells [ 26, 28– 33].