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To move beyond this stage, the development of well-functioning clusters is essential.
Understanding the mechanisms that govern energy transport within clusters is essential to optimizing their structure and composition for eventual development of QD-included materials with long-term photostability.
Generation of correct clusters is essential for the correct determination of gene ancestry, but it is not the sole limiting factor.
An extended chain of Fe S (iron sulfur) clusters is essential in transferring electrons from the flavin to the quinone-binding site [ 2– 4].
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Natsuaki Y, Egawa G, Nakamizo S, et al. Perivascular leukocyte clusters are essential for efficient activation of effector T cells in the skin.
Iron sulfur (Fe-S) clusters are essential bioinorganic factors that facilitate mitochondrial metabolism.
Iron sulfur (Fe-S) clusters are essential for mitochondrial metabolism, but their regulation in pulmonary hypertension (PH) remains enigmatic.
Heme and iron-sulfur (Fe-S) clusters are essential for the function of ROS-producing enzymes such as NADPH and oxidases involved in mitochondrial function.
Because Fe-S clusters are essential protein cofactors involved in many cellular pathways (Lill, 2009), abnormal or loss of frataxin function can cause multiple and cumulative cellular dysregulations.
Fe-S clusters are essential prosthetic groups for many proteins with a variety of functions and subcellular localisations (Lill, 2009; Ye and Rouault, 2010).
For all these functions, its two Fe-S clusters are essential (Kispal et al., 2005; Khoshnevis et al., 2010; Barthelme et al., 2011; Becker et al., 2012).
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