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The fact that productions form clusters could also indicate discreteness, as long as each cluster of productions was completely different from the others.
Many other small meta-clusters, gathering less than 10 clusters, could also be identified but will not be discussed in the present study.
Combining all of the nodes in the detected clusters could also further improve the multilevel layout solutions, especially in networks such as the AP/MS-Combined, which show exceptionally high and extensive clustering structure.
However, while clustering of persistent gene provided a significant opportunity to allow the genes to be inherited and spread, these clusters could also be destroyed by inevitable random gene insertion.
Regular translocations between areas within clusters could also be supported in order to maintain the genetic diversity [ 15, 20], particularly for entirely or partially fenced protected areas (e.g. Malilangwe, Hluhluwe-iMfolozi, etc).
In addition, the genes in these clusters could also act as "helper NB-LRRs", mediating signal transduction downstream of various different NB-LRR receptors for activation during effector-triggered immunity, thereby leveraging functional constraints as previously made evident for ADR1 family in A. thaliana[ 91, 92].
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If the spatial arrangement were specified, the cluster could also be interpreted as a plot with m sub-plots.
Furthermore, no Er3+ clusters are found in all the films so that the quenching phenomenon caused by Er3+ clustering could also be disregarded [15].
In contrast, sibling clustering could also relate to recessively inherited susceptibility.
Thus, we reason that mutations in the rest of the genes within the CPS cluster could also cause translucent morphology.
This suggests that the miR379 410 cluster could also be involved in the Mef2-dependent control of synapse number at later stages of neuronal development.
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